custom 180 k oligonucleotide microarray Search Results


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WholeGenome LLC 4×180k wholegenome oligonucleotide microarray
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CapitalBio Corporation lncrna chip
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Oxford Gene Technology custom designed 4 × 180k oligonucleotide microarray
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Oxford Gene Technology 180k-feature whole-genome microarray cytosure iscav2
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Oxford Gene Technology 4 × 180 k microarrays cytosure isca v3
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CapitalBio Corporation microarray v4.0
Relative expression of lncRNAs. ( A, B ) The raw images of the <t>microarray</t> analysis were shown; ( C ) the scatter plot was shown; ( D ) The comparison of relative expression of NRAD1 in HCC and normal cell lines; ( E ) The comparison of relative expression of LINC00152 in HCC and normal cell lines.
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CombiMatrix 180k oligonucleotide microarray
Case #2's CGH analysis (DNAarrayTM-Oligi-180) revealed ( A ) a single copy number decrease of 3.0 Mb of chromosome 17p13.3 region and ( B ) a single copy increase of 2.0 Mb of chromosome 16p13.3 region consistent with a 17p13.3 microdeletion and a 16p13.3 microduplication. The derivative chromosome likely represents a malsegregation of a paternal balanced chromosome 16;17 translocation. The 16p duplication does not encompass the CREBBP gene within the Rubenstein–Taybi critical region. CGH, <t>comparative</t> genomic <t>hybridization;</t> CREBBP, CREB binding protein (CREB, cAMP response element binding protein binding protein).
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Image Search Results


Relative expression of lncRNAs. ( A, B ) The raw images of the microarray analysis were shown; ( C ) the scatter plot was shown; ( D ) The comparison of relative expression of NRAD1 in HCC and normal cell lines; ( E ) The comparison of relative expression of LINC00152 in HCC and normal cell lines.

Journal: OncoTargets and therapy

Article Title: Long Non-Coding RNA NRAD1 and LINC00152 are Highly Expressed and Associated with Prognosis in Patients with Hepatocellular Carcinoma

doi: 10.2147/OTT.S251231

Figure Lengend Snippet: Relative expression of lncRNAs. ( A, B ) The raw images of the microarray analysis were shown; ( C ) the scatter plot was shown; ( D ) The comparison of relative expression of NRAD1 in HCC and normal cell lines; ( E ) The comparison of relative expression of LINC00152 in HCC and normal cell lines.

Article Snippet: We performed LncRNA and mRNA human gene expression microarray V4.0 (Capitalbio, 4×180K, two-channel, containing about 37 thousand lncRNAs and 34 thousand mRNAs) was applied to the profiling of lncRNAs in four cancerous tissues and the paired paracancerous tissues.

Techniques: Expressing, Microarray, Comparison

Case #2's CGH analysis (DNAarrayTM-Oligi-180) revealed ( A ) a single copy number decrease of 3.0 Mb of chromosome 17p13.3 region and ( B ) a single copy increase of 2.0 Mb of chromosome 16p13.3 region consistent with a 17p13.3 microdeletion and a 16p13.3 microduplication. The derivative chromosome likely represents a malsegregation of a paternal balanced chromosome 16;17 translocation. The 16p duplication does not encompass the CREBBP gene within the Rubenstein–Taybi critical region. CGH, comparative genomic hybridization; CREBBP, CREB binding protein (CREB, cAMP response element binding protein binding protein).

Journal: Journal of Pediatric Genetics

Article Title: Case Report of Proliferative Peripheral Retinopathy in Two Familial Lissencephaly Infants with Miller–Dieker Syndrome

doi: 10.1055/s-0037-1612638

Figure Lengend Snippet: Case #2's CGH analysis (DNAarrayTM-Oligi-180) revealed ( A ) a single copy number decrease of 3.0 Mb of chromosome 17p13.3 region and ( B ) a single copy increase of 2.0 Mb of chromosome 16p13.3 region consistent with a 17p13.3 microdeletion and a 16p13.3 microduplication. The derivative chromosome likely represents a malsegregation of a paternal balanced chromosome 16;17 translocation. The 16p duplication does not encompass the CREBBP gene within the Rubenstein–Taybi critical region. CGH, comparative genomic hybridization; CREBBP, CREB binding protein (CREB, cAMP response element binding protein binding protein).

Article Snippet: Comparative genomic hybridization (CGH; 180K oligonucleotide microarray; DNAarray-Oligo 180K, CombiMATRIX Molecular Diagnostics, Inc., Irvine, California, United States) revealed an abnormal male genotype, a terminal 3.0 Mb microdeletion in short arm of chromosome 17 (17p13.3) encompassing the PAFAH1B1 and YWHAE genes, a 2.0 Mb microduplication in chromosome 16p13.3, a common variant pericentric inversion in chromosome 9 (9p11q13), and several chromosomal polymorphisms (not shown).

Techniques: Translocation Assay, Hybridization, Binding Assay, Protein Binding